Pneumonia (ventilator-associated or hospital-acquired)
Overall Bottom Line
- Clinical criteria for ventilator-associated pneumonia (VAP) include new lung infiltrate in a ventilated patient and at least two of the following: temperature >38°C, leukopenia or leukocytosis, and purulent secretions. C Semi-quantitative culture is useful for the identifying causative organism; sampling by endotrachial aspiration is recommended for patients with VAP and using spontaneous sputum, induced sputum, or nasotracheal suctioning in non-ventilated patients with HAP.C
- Antimicrobial therapy should be started promptly and based on local resistance patterns. The decision regarding single, dual or triple antibiotic therapy depends on patient mortality risk, risk factors for MDR pathogens, and whether HAP or VAP is treated. 27B
- The recommended duration is 7 days for patients with HAP or VAP who have a good clinical response. 27B
- Measures to prevent VAP include a bundle of services: oral decontamination, elevating head of bed to 30° to 45°, use of a new ventilator circuit for each patient and replacing it when soiled, suction of subglottic secretion, and minimizing the duration of intubation. A
Background
Ventilator-associated pneumonia (VAP) is pneumonia that occur 48 to 72 hours after endotracheal intubation. Early onset VAP occur within 4 days of intubation, while late onset VAP occur 5 days or more after intubation. Hospital-acquired pneumonia (HAP) is pneumonia that begins after hospitalization but is not associated with mechanical ventilation.Nosocomial pneumonia encompasses both HAP and VAP.
Incidence
Other Impact
Causes of the Condition
- VAP is caused by a wide spectrum of organisms.
- The most common organisms causing nosocomial pneumonia are KEEPS: klebsiella, enterobacter, e.coli, pseudomonas aeurignosa, and staphylococcus aureus.
- Common bacteria include aerobic Gram-negative bacilli such as Pseudomonas aeruginosa, Escherichiacoli, Klebsiella pneumoniae, and Acinetobacter species.
- Infections due to Gram-postive cocci such as Staphylococcus aureus including MRSA are increasing.
Pathophysiology
- Most cases of VAP are caused by the aspiration of infected secretion from oropharynx, although a few cases occur secondary to blood stream infection.
- Critical illness leads to rapid colonization of the oropharynx with pathogenic bacteria, caused by changes in the host defense, antibiotic exposure, and possible changes in bacterial adhesion and host surface receptors.
- Many of the usual host defense mechanisms including immune function are less effective.
- Profound changes occur in the function and structure of the alveoli.
- Neutrophil dysfunction and inadequate phagocytosis increase susceptibility to infection.
Risk Factors
Risk Factor |
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Intubation duration |
Mechanical ventilation duration |
Colonization of the ventilator circuit |
Supine body position |
Enteral nutrition |
Oropharyngeal colonization |
Systemic antibiotics for prolong period |
H2 blockers and antacids |
Blood transfusion |
Hyperglycemia |
Cigarette smoking |
Male sex |
SOFA score |
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